How to read, respond to, and act decisively on life-threatening rhythms
Why Rhythm Recognition Drives ACLS Success
Cardiac arrest rhythms are more than abstract waveform patterns—they are time-sensitive clinical situations demanding confident action. ACLS-trained providers must translate ECGs into treatment decisions within seconds. Each delay costs myocardial viability and neurologic recovery potential.
Correct rhythm identification means the difference between a defibrillation shock and a medication push. It determines whether compressions continue or an airway intervention takes priority. Advanced ECG recognition goes beyond strip memorization. Providers must interpret rhythm origin, stability, and rate in real time while under pressure.
Mastering this skill directly supports the ACLS certification training at EMS Ricky. It also builds the foundation for successful resuscitation outcomes and seamless teamwork.
Differentiating Shockable and Non-Shockable Rhythms
Ventricular Fibrillation (VF)
VF appears as a chaotic, disorganized waveform with no discernible P, QRS, or T complexes. Coarse VF may show high-amplitude waves, while fine VF looks nearly flat. Neither rhythm produces a pulse.
In ACLS, VF is always treated with immediate defibrillation. Delayed shocks reduce the chance of survival with each passing minute. Compressions resume immediately after the shock, without waiting for rhythm confirmation.
Pulseless Ventricular Tachycardia (VT)
Pulseless VT presents as regular, wide QRS complexes often exceeding 100 beats per minute. Despite organized electrical activity, there is no effective cardiac output.
This rhythm also qualifies for shock treatment. Defibrillation remains the first step, followed by high-quality CPR. Providers should prepare antiarrhythmic medications and recheck the rhythm after two-minute cycles.
Asystole and Pulseless Electrical Activity (PEA)
Asystole is a true flatline—no ventricular activity, no pulse. PEA features organized electrical activity on the monitor without a palpable pulse.
Neither rhythm benefits from defibrillation. Instead, the focus turns to high-quality CPR, epinephrine administration, and identifying reversible causes (the Hs and Ts). Quick identification of the correct rhythm category ensures appropriate use of resources and time.
Recognizing which rhythms require defibrillation versus medication is critical to ACLS success. The table below outlines how to categorize common cardiac arrest rhythms and what actions to prioritize first.
Rhythm Type | Shockable? | First-Line Actions |
---|---|---|
Ventricular Fibrillation (VF) | Yes | Immediate defibrillation, CPR, epinephrine, then amiodarone if refractory |
Pulseless Ventricular Tachycardia (VT) | Yes | Immediate defibrillation, CPR, epinephrine, antiarrhythmic if needed |
Asystole | No | CPR, epinephrine every 3–5 minutes, evaluate Hs and Ts |
Pulseless Electrical Activity (PEA) | No | CPR, epinephrine, identify and correct reversible causes |
Monomorphic VT (with pulse) | Conditionally | Assess for stability; cardiovert if unstable, antiarrhythmic if stable |
Supraventricular Tachycardia (SVT) | No | Vagal maneuvers, adenosine (if regular), monitor stability |
When to Shock, Push Drugs, or Continue CPR

The ACLS Code Clock
During a code, timing determines treatment. Every two-minute CPR cycle resets the clock for rhythm checks and interventions. Defibrillation and medications must align with this rhythm.
Epinephrine is administered every 3 to 5 minutes. This usually translates to once every other CPR cycle. Shockable rhythms are evaluated and treated with defibrillation at the beginning of a cycle, followed by compressions.
Delays between rhythm recognition and treatment cause confusion and error. Teams must internalize the two-minute rhythm for synchronized care delivery.
Antiarrhythmics in ACLS: Amiodarone vs. Lidocaine
Antiarrhythmic drugs play a role when defibrillation and CPR do not restore a perfusing rhythm. Amiodarone is the preferred first-line agent, with lidocaine as a secondary option or substitute.
The initial amiodarone dose is 300 mg IV push after the third shock, followed by 150 mg if needed. Lidocaine dosing begins with 1 to 1.5 mg/kg IV and continues in divided doses up to 3 mg/kg.
Post-resuscitation, these agents may continue as maintenance infusions to prevent recurrence.
ACLS Antiarrhythmic Dosing Reference Table
Medication | Initial Dose | Repeat Dose | Max Dose | Infusion Maintenance |
---|---|---|---|---|
Amiodarone | 300 mg IV push | 150 mg IV push | 450 mg total | 1 mg/min for 6 hrs, then 0.5 mg/min |
Lidocaine | 1-1.5 mg/kg IV | 0.5-0.75 mg/kg q5-10m | 3 mg/kg | 1-4 mg/min continuous |
Managing Tachycardia with a Pulse
Regular vs Irregular Rhythms
Stable tachycardias demand rhythm analysis. A regular rhythm suggests SVT or monomorphic VT. Irregular rhythms suggest atrial fibrillation or polymorphic VT.
QRS width further guides treatment. Narrow complexes point to supraventricular origins. Wide complexes suggest ventricular or aberrant conduction. ECG precision ensures appropriate medication and cardioversion decisions.
Stable vs. Unstable Patient Decision Tree
The clinical picture must drive intervention choice. Unstable patients show signs of poor perfusion: hypotension, altered mentation, or chest pain. In these cases, synchronized cardioversion is immediately warranted.
Stable patients allow more cautious rhythm identification and pharmaceutical interventions. Misjudging this distinction risks progression to arrest or incorrect treatment.
ACLS Drugs for Tachycardia
Adenosine is the agent of choice for regular, narrow-complex tachycardias. A rapid 6 mg IV push is followed by 12 mg if necessary. Providers should monitor for transient asystole during conversion.
Wide QRS tachycardias may benefit from amiodarone, procainamide, or sotalol. Each requires slower administration and monitoring for QRS prolongation or hypotension.
Critical ECG Features for Fast Identification
Wide vs. Narrow QRS
A QRS complex over 0.12 seconds indicates a wide-complex rhythm. These often stem from ventricular origin or conduction abnormalities. Wide complexes suggest greater instability and may warrant synchronized cardioversion.
Narrow complexes originate above the ventricles. While potentially fast and symptomatic, they often respond to vagal maneuvers or adenosine.
Regular vs. Irregular Rhythms
Irregular rhythms pose more diagnostic challenge. Atrial fibrillation lacks P waves and produces an irregularly irregular QRS cadence. Irregular rhythms require special caution with AV nodal blocking agents.
Regularity helps rule in or out reentrant SVT, flutter, or monomorphic VT.
Monomorphic vs. Polymorphic Tachycardia
Monomorphic VT shows a consistent QRS shape and rate. Polymorphic VT, including torsades de pointes, features QRS complexes that vary in shape and amplitude.
Torsades requires treatment with magnesium sulfate and correction of underlying causes like hypokalemia or prolonged QT. Immediate recognition enables life-saving interventions.
Decision-Making During Code Scenarios
Two-Minute Rhythm Rechecks — Avoiding Delays
Rhythm checks should not interrupt compressions for more than 10 seconds. Teams must prepare in advance and coordinate clearly to minimize pauses.
Rhythm evaluation must follow each two-minute cycle. Pauses longer than 10 seconds reduce coronary perfusion pressure and lower ROSC chances. Rapid defibrillator application and team preparation keep delays minimal.
Hs and Ts — Diagnosing the Underlying Cause
Unresponsive rhythms may result from correctable conditions. The ACLS mnemonic Hs and Ts covers common culprits:
Hs: Hypoxia, hypovolemia, hydrogen ion (acidosis), hypo/hyperkalemia, hypothermia
Ts: Tension pneumothorax, tamponade, toxins, thrombosis (pulmonary or coronary)
Identifying and reversing these factors often makes the difference between failed and successful resuscitation.
Rhythm Management After ROSC (Return of Spontaneous Circulation)
Monitoring for Recurrent VF or VT
After ROSC, continuous cardiac monitoring is essential. Recurrent ventricular dysrhythmias can occur without warning. A well-prepared provider anticipates re-arrest and keeps medications and defibrillator ready.
Antiarrhythmic infusions may continue for hours post-code. ECG changes, vital signs, and underlying causes all require close observation.
Preventing Secondary Cardiac Arrest
Post-arrest care prevents recurrence and supports recovery. Treating ischemia, maintaining normoxia, and avoiding hypotension support cardiac function.
Sedation, cooling protocols, and fluid management also play a role. Providers must work alongside ICU and transport teams to ensure continuity. Interfacility transitions often require precise drug handoffs and rhythm reports.
What Every ACLS Provider Should Master by Heart
ACLS rhythm management requires more than algorithm recall. The provider must link rhythm interpretation with cause identification and timely intervention.
Each ECG pattern demands a specific response. Confident providers act early, direct clearly, and maintain hands-on involvement throughout the code. The tools taught in ACLS certification are only useful when applied with clinical judgment.
Ready to build your rhythm confidence? Get started by exploring the application process at EMS Ricky and bring clinical meaning to your ECGs.